is the form when the affected has a complete loss of the enzyme. Both parents must contribute a galactosemic gene for a child to receive this very deadly form of the disorder. Because the body has none of the enzyme needed to turn galactose into glucose an accumulation occurs that is like a poison and is highly fatal. Classic Galactosemia is treatable through a strict diet of absolutely no lactose or galactose. Some common foods containing these are dairy products‚ legumes‚ breast milk‚
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is burned? Ash 9. How much percentage of a person’s body weight is protein? Approx 15% 10. What is the proper terminology for “blood sugar”? Glucose 11. Why is fiber an essential part of one’s diet? It adds volume which fills the stomach which contributes to a full feeling so eating stops 12. Where are the following monosaccharides found: a. Glucose In the blood b. Fructose Fruits c. Galactose Milk 13.
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solution. In this experiment‚ all the test solutions show a positive test with Molisch reagent and concentrated sulfuric acid to form a violet ring at the interface of two layers. This is because the samples used in this experiment are maltose‚ glucose and starch which are considered as carbohydrate that should give a positive reaction. Monosaccharides give a rapid positive test compare to disaccharides and polysaccharides which will react slower. Therefore‚ the
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for the unknown solution is‚ #128. Results The Results of Each Solution When Mixed with Each Type of Tests Test Tube Samples | Benedict Before After | Biuret | Iodine | #1. 1% glucose solution | light blue | red/brown | light blue | pale yellow | #2. 0.3% glucose - 1- phosphate | light blue | no colour change | light blue | pale yellow | #3. 1% maltose solution | light blue | red/brown | light blue | pale yellow | #4. honey solution | light blue | orange/red | light
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Hypoglycemia is a condition characterized by an abnormally low level of blood sugar (glucose) in the body. It is most commonly associated with diabetes‚ however there are a small amount of conditions in which hypoglycemics have never experienced any form of diabetes. Basically‚ hypoglycemia is the body ’s inability to properly handle the large amounts of sugar that are usually present in the typical North American diet. Non diabetic hypoglycemia is characterized typically by the over production of
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Negative result: No precipitate Seliwanoff’s Test Tests for: Fructose (+)‚ Other (–) Positive result: Change in color to orange Negative result: Not orange Glucose Oxidase Test Tests for: Glucose (+)‚ Other (–) Positive
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BI108 QUIZ No2: Chapters 3 & 4: Macromolecules 1. . The major carbohydrate-storage molecule in plants is: a. starch. b. cellulose. c. glycogen. d. deoxyribonucleic acid. e. maltose 2. The helix is an example of which level of protein structure? a. Primary structure b. Quaternary structure c. Secondary structure d. Tertiary structure e. none of the above 3. The number of D-amino acids that occur naturally in proteins is: a. zero. b. 20. c. 19. d. 9. e. none of the above 4. In polysaccharides
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submission: Date of Experiment: Introduction Glucose-6-phosphase (G6Pase) is an enzyme presented in liver and used in the hydrolysis of glucose-6-phosphate (G6P). This catalyst the final step in glycogeneogeneis and glycogenolysis which is the key point of homeostatic regulating the blood glucose levels in our bodies. In the experiment‚ we will determine the activity of G6Pase. Frist‚ we will extract the G6Pase from
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measuring cylinder PROCEDURE PART A:TEST FOR DETECTING CARBOHYDRATES 1. MOLISCH TEST a)Small paper labels‚number five clean test tubes are using as directed below: i. 20 drops of deionized water(control) ii.20 drops of 1% glucose solution(1g of glucose is added in 99ml distilled water) iii.20 drops of 1% sucrose solution iv.20 drops of 1% lactose solution v.20 drops of 1% starch solution(1g of starch is added in 99ml distilled water and put in boil) b) Add 3 drops of Molisch
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1. Introduction People still die from diabetic ketoacidosis. Poor patient education is probably the most important determinant of the incidence of the catastrophe that constitutes "DKA". In several series‚ only about a fifth of patients with DKA are first-time presenters with recently acquired Type I diabetes mellitus. The remainder are recognised diabetics who are either noncompliant with insulin therapy‚ or have serious underlying illess that precipitates DKA. Most such
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