It has been debated whether children who develop Schizophrenia have a notable genetic connection or not. Researchers question whether the disease is passed down in familial lines or it is just triggered by an external source, but there is data that show genetic abnormalities in patients who develop childhood-onset Schizophrenia. In a study by C.J. Carter, he concluded “genes related to glutamatergic and in particular NMDA receptor signaling” have been associated with Schizophrenia, the specific genes Carter mentions are “DAOBS, DAOABS, GRIN1BS, GRIN2ABS, GRIN2BBS, GRIN2DS, PPP3CCS, NOS1BS, NOSIAPB,” (Carter). Carter’s study did find altered genes in individuals who developed Schizophrenia, but how strong is the relation between these genes and the development of the disorder. Carter himself even stated that “although many genes have been associated with [Schizophrenia], there is extreme disparity between the individual gene association results,” (Carter). Genetic connections to the disease can be found, but how valid is the connection? Primarily each person has 46 chromosomes, and each chromosome has upwards of fifteen thousand genes- that’s …show more content…
There are other environmental factors that can be related to genetic factors that have a connection to Schizophrenia. One example could be “the association of APOEBS with Schizophrenia,” an occurrence in the Chinese population was seen during times of famine when more babies were born with this gene and later developed Schizophrenia (Carter). This association does imply the nature and nurture based development of Schizophrenia because without the famine the APOEBS gene most likely would not have been present in the babies and in all probability the babies would not have developed Schizophrenia later on in