Nt1310 Unit 3 Lab Report
. Na+ and K+. K+ was more concentrated inside the cell while Na+ was more concentrated outside the cell.
2. A cell is at rest without any implications of changing the potential.
3.
4.
5. K+ is more permeable than Na+ in a resting neuron. This is because there are few sodium leakage channels.
6. Since the resting membrane potential is the potential difference between the inside and outside of the cell across the membrane, a change would depend on the resting permeability of the membrane to ions.
Activity #2
1.
2. Graded potentials can be said to be momentary, localized changes in the membrane potential that can be either depolarizing or hyperpolarizing.
3. In the Pacinian corpuscle, the moderate intensity pressure modality induced a receptor potential of the …show more content…
This means that action potential occurs only once threshold is met.
5. The trigger zone.
Activity #4
1. TTX blocks the diffusion of sodium through the voltage-gated Na+ channels.
2. Lidocaine blocks the diffusion of Na+ through the voltage-gated channels.
3. With a compound action potential, the results would not necessarily be zero because some axons could remain unaffected.
4. TTX blocked the Na+ channels which prevented the propagation of the action potential from R1 to R2.
5. Lidocaine blocks the Na+ channels which prevents the propagation of the action potential from R1 to R2.
6. Lidocaine should be injected to the receptors to prevent the generation of an action potential that would lead to the perception of pain.
Activity #5
1. Inactivation is the voltage-gated sodium channels are inactive once they stop allowing Na+ to pass through.
2. It is the period of when there is zero action potential that can be generated regardless of the strength of the stimulus.
3. The threshold for the second action potential increased as the interval between the stimuli decreased as predicted.
4. A greater stimulus is required because voltage-gated K+ channels that oppose depolarization are open during this
2. A cell is at rest without any implications of changing the potential.
3.
4.
5. K+ is more permeable than Na+ in a resting neuron. This is because there are few sodium leakage channels.
6. Since the resting membrane potential is the potential difference between the inside and outside of the cell across the membrane, a change would depend on the resting permeability of the membrane to ions.
Activity #2
1.
2. Graded potentials can be said to be momentary, localized changes in the membrane potential that can be either depolarizing or hyperpolarizing.
3. In the Pacinian corpuscle, the moderate intensity pressure modality induced a receptor potential of the …show more content…
This means that action potential occurs only once threshold is met.
5. The trigger zone.
Activity #4
1. TTX blocks the diffusion of sodium through the voltage-gated Na+ channels.
2. Lidocaine blocks the diffusion of Na+ through the voltage-gated channels.
3. With a compound action potential, the results would not necessarily be zero because some axons could remain unaffected.
4. TTX blocked the Na+ channels which prevented the propagation of the action potential from R1 to R2.
5. Lidocaine blocks the Na+ channels which prevents the propagation of the action potential from R1 to R2.
6. Lidocaine should be injected to the receptors to prevent the generation of an action potential that would lead to the perception of pain.
Activity #5
1. Inactivation is the voltage-gated sodium channels are inactive once they stop allowing Na+ to pass through.
2. It is the period of when there is zero action potential that can be generated regardless of the strength of the stimulus.
3. The threshold for the second action potential increased as the interval between the stimuli decreased as predicted.
4. A greater stimulus is required because voltage-gated K+ channels that oppose depolarization are open during this