Preview

Metabolic Acidosis Research Paper

Good Essays
Open Document
Open Document
764 Words
Grammar
Grammar
Plagiarism
Plagiarism
Writing
Writing
Score
Score
Metabolic Acidosis Research Paper
Metabolic Acidosis

Metabolic acidosis is a condition in which there is too much acid in the blood. It happens because of a chemical imbalance in your cells. Metabolic acidosis can happen at any age, and there are many different causes. It may be a symptom of a sudden, short-lived (acute) condition, or a lifelong (chronic) condition.
Metabolic acidosis can be mild, or it can be severe and life-threatening, depending on the cause. It can be corrected if the cause is identified and treated correctly.

CAUSES
There are many possible causes of metabolic acidosis. The most common causes are:
• The body producing too much acid.
• Losing too much of a chemical that balances acid levels (bicarbonate). This can happen because of diarrhea or
…show more content…
Do not stop taking the antibiotic even if you start to feel better.
• Drink enough fluid to keep your urine clear or pale yellow.
• Follow instructions from your health care provider about eating or drinking restrictions.
• Pay attention to your symptoms and any changes in your symptoms.
• Keep all follow-up visits as told by your health care provider. This is important.

PREVENTION
Take these actions to prevent metabolic acidosis:
• Manage any chronic conditions you have.
• Avoid exposure to toxins.
• Avoid drinking excessive amounts of alcohol.
• Understand your medicines and any supplements you take, and their possible side effects.
• Watch for any symptoms of metabolic acidosis to develop.

SEEK MEDICAL CARE IF:
• You have new symptoms.
• You have side effects from medicines you are taking.
• You have nausea, vomiting, or diarrhea that does not get better with medicine or gets worse.
• You have body aches or lethargy that gets worse.
• You have symptoms of dehydration, such as dark urine or decreased frequency of urination.
• You have a decreased appetite.
• You have a

You May Also Find These Documents Helpful

  • Good Essays

    21-year old woman that has had type 1 diabetes for the past 8 years, was brought to the hospital in a coma. She was prescribed to take 92 units of insulin a day to maintain her sugar levels within normal limits and prevent excess sugar in her urine. Upon admission she was hypontensive, tachycardic and hyperventilating. Her labs show she is acidonic, arterial blood carbon dioxide levels were low, blood oxygen tension is normal, bicarbonate levels are really low indicating metabolic acidosis, low sodium levels, slightly high level of potassium, Chloride level is on the low end of normal, very high levels of blood urea and nitrogen, total carbon dioxide levels are really low, extremely high sugar…

    • 681 Words
    • 3 Pages
    Good Essays
  • Satisfactory Essays

    1. Rebreathing simulates You correctly answered: a. hypoventilation and respiratory acidosis. 2. Hypoventilation results in You correctly answered: c. an accumulation of carbon dioxide in the blood. 3. The renal system can compensate for respiratory acidosis by You correctly answered: b. excreting H+ and retaining bicarbonate ion. 4. Respiratory acidosis can be caused by all of the following except You correctly answered: d. an anxiety attack.…

    • 387 Words
    • 2 Pages
    Satisfactory Essays
  • Satisfactory Essays

    Potential Causes: COPD plus DKA or renal failure, any combination of things that cause each one…

    • 457 Words
    • 1 Page
    Satisfactory Essays
  • Good Essays

    Wgu Est1 Task 4

    • 985 Words
    • 4 Pages

    Aldolase B, can no longer take its substrate, F-1-P, and turn it into the products, DHAP and Glyceraldehyde. During HFI, fructose is still being phosphorylated by fructokinase, leading to a build up of F-1-P, and will no longer being used for glycolysis or gluconeogenesis. The continued use of phosphorous leads to the depletion of the free phosphate pool in the cells. The electron transport chain requires phosphate to make ATP. With the low amounts of free phosphate available, ATP production slows. Essentially, fructose is no longer being used as energy by the liver cells. Liver cells are now low on energy leading to liver damage and eventually liver failure. Fructose-1-phosphate produces the symptoms of HFI. It normally acts a signal in high blood sugar instructing the glucokinase to stay in the cytoplasm, so it does not go into the nucleus. When blood sugar is low, and F-1-P builds up, it signals the glucokinse to stay in the cytoplasm leading to a glycogenolysis and gluconeogenesis slowing down. When low blood sugar occurs, the liver cannot release glucose into the blood to help stabilize it, this is known as hypoglycemia. Many symptoms that are involved with hereditary fructose intolerance have to do with hypoglycemia, such as shakiness, headaches, and irritability, in addition to phosphate related liver issues (Sanders,…

    • 985 Words
    • 4 Pages
    Good Essays
  • Good Essays

    Chapter 8 Quizzes

    • 733 Words
    • 3 Pages

    The patient has acidosis that might be respiratory in origin. Which of the following is the major cause of acute primary respiratory acidosis?…

    • 733 Words
    • 3 Pages
    Good Essays
  • Satisfactory Essays

    (vomiting and diarrhea at the same time, majority of the people die in 7 days, rapid dehydration)…

    • 283 Words
    • 2 Pages
    Satisfactory Essays
  • Good Essays

    Aspirin Research Paper

    • 1199 Words
    • 5 Pages

    In majority of cases, some degree of acid-base disturbances exist. A mixed respiratory alkalosis and metabolic acidosis with either normal or high arterial pH (normal or decreased hydrogen ion conc.) is usual in adults and children ages 4 years or over. In children aged under 4 years, a dominant metabolic acidosis with low arterial pH (raised hydrogen ion concentration) is common. Increased acidity in the blood, may increase salicylate transfer across the blood brain barrier.…

    • 1199 Words
    • 5 Pages
    Good Essays
  • Good Essays

    Answers to work sheet

    • 581 Words
    • 3 Pages

    Why does potassium concentration rise in patients with acidosis? What is this called? What effects does it have?…

    • 581 Words
    • 3 Pages
    Good Essays
  • Satisfactory Essays

    Endocrine

    • 404 Words
    • 2 Pages

    A patient in DKA needs more insulin to get the glucose into the cells. Since this isn't happening they body is using fats for fuel instead. Now they have…

    • 404 Words
    • 2 Pages
    Satisfactory Essays
  • Good Essays

    Atkins Diet Analysis

    • 425 Words
    • 2 Pages

    Her bowel sounds were extremely active, some pain in the upper-central abdomen, and a urine analysis which confirmed ketonuria or for the abnormal amount of ketones. As a result, she was sent to the intensive care unit for metabolic acidosis or when the body makes too much acid or when the kidneys are not sufficient when removing acid from the body. There are four things that are when ingested, are known to cause metabolic acidosis: methanol, ethylene glycol, L- or D-lactate acidosis, or ketoacidosis (from diabetes mellitus, alcohol, or starvation). Doctors found that she did not ingest any of these things, but discovered high levels of beta hydroxy acids and keto acids which is usually found in ketoacidosis. Her liver was producing too many ketones because her liver detected a severe decrease in insulin characteristic to starvation. The Atkins diet is aware of ketonuria, but similar low carbohydrate diets such as Atkins are known to increase the production of ketones. The stress from the starvation made her ketonuria worsen.…

    • 425 Words
    • 2 Pages
    Good Essays
  • Good Essays

    Lactic acidosis (too much lactate in the blood and low blood pH – occurs more frequently with renal insufficiency…

    • 2421 Words
    • 10 Pages
    Good Essays
  • Powerful Essays

    Hypovolemic Shock

    • 1713 Words
    • 5 Pages

    In the initial stage signs of shock are minimal with cells switching from aerobic to anaerobic metabolism leading to lactic acidosis, this is the build-up of excess lactic acid in the blood. Taking an arterial blood gas and checking the lactate level in the blood can give clear indication of acidosis. Hypovolemia can manifest when there is about 15% loss of circulatory volume, but the more volume lost the more significant the clinical signs become (Hall, 2010).…

    • 1713 Words
    • 5 Pages
    Powerful Essays
  • Good Essays

    The signs of dehydration are often noticeable. Dry, cracking lips and thirst are usually the first symptoms of dehydration. Then there is little or no urine, or darkened urine. Eventually, there will be dizziness, fatigue, and headaches. Those suffering from more severe dehydration face worse than discomfort. Lack of water can result in rapid heart rate, fevers, shock, and seizures. If a person faints, has difficulty breathing, or feels chest or abdominal pain, they must be taken to a hospital…

    • 416 Words
    • 2 Pages
    Good Essays
  • Good Essays

    Metabolic acidosis due to low serum bicarbonate and low pH from the body’s metabolic process…

    • 469 Words
    • 2 Pages
    Good Essays
  • Satisfactory Essays

    Alcohol Q/A

    • 621 Words
    • 2 Pages

    Me being a teenager who would never dare to drink I would have to say i guess completely abstain from drinking or drink only one or two glasses very slowly.…

    • 621 Words
    • 2 Pages
    Satisfactory Essays