Acute Myeloid Leukemia

Leukemia is the tenth most prevalent cancer found in humans. The most common type of leukemia is acute myeloid leukemia. This type of leukemia begins in immature myeloid cells: white blood cells, red blood cells, or cells that make platelets known as megakaryocytes. Acute myelogenous leukemia is different from chronic myelogenous leukemia in that it spreads quicker and is much more deadly than the latter. Acute myeloid leukemia is characterized by the location of development, in stem cells, which are immature, also called “blasts.” Chronic leukemias form in the actual cell, after it has formed from a stem cell. AML does not divide more rapidly than normal cells, but it does not hit the checkpoint in which it is to stop dividing. It continues to divide.
Leukemia itself was first discovered in 1827 by the French physician Alfred Armand Louis Marie Velpeau. He documented the case of one of his patients, who suffered from a disease that came with fever, chills, and blood with “the consistency of gruel.” Later, the term leucocythemia was adopted in 1845, until a German pathologist named Virchow introduced “Leukemia,’ which is Greek for “white blood.” In 1856. Rudolf Virchow is
Smoking, long term exposure to the chemical benzene, radiation exposure, certain chemotherapeutic drugs, and a history of blood disorders can all increase the risk of developing AML. The average age for acute myelogenous leukemia is 66, and it is more prominent in males. Having specified genetic syndromes such as Down syndrome can also increase the risk of acute myeloid leukemia. These are only factors, and do not mean that everyone over the age of sixty will have AML, or every smoker will develop the disease. It can happen at any age, and any state of health. Each cell in the body has genes that work as overseers of a sort, telling the cell what to do. When one of these genes has a mutation, that’s when AML