Pain Management and Quality of Life for Sickle Cell Disease Patients Cynthia Evans Mississippi College Pain Management and Quality of Life for Sickle Cell Disease Patients This is a review of literature examining how frequent recurring episodes of pain affect the quality of life in the sickle cell disease patients. Several studies conducted concerning pain management and quality of life for sickle cell disease patients indicate additional studies needed. Identifying additional effective
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Genetics of Sickle Cell Anemia Objectives • To observe how a disease can act as a selective force • To describe changes in allele frequencies in a population as a result of a selective force Hypothesis The starting ratio of HbA to HbS is 3:1; in a typical population‚ the Homozygous Dominant Gene would become the standard‚ but because carrying the Heterozygous version of the allele has an advantage to fighting Malaria it will become the dominant Allele for the population. The HbA/HbS gene will
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Sickle Cell Plan of Care Read the situation provided. Then‚ provide a brief description of the pathophysiology of sickle cell anemia and complete the nursing care plan by filling in the goals‚ outcomes‚ and nursing orders for the diagnoses provided in the table. SITUATION: Lavon is a 30 year old‚ single African American who was diagnosed with sickle cell anemia when he was 4 years old. He works for a computer company and has been working 12 hour days to meet the deadline for a special project
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Sickle Cell Anemia "An ISU student died March 12 from complications of Sickle Cell Anemia there is no cure for Sickle Cell Anemia" (Indiana Statesman‚ 2004). What is Sickle Cell Anemia? According to National Institute of Health‚ Sickle Cell Anemia is a lifelong‚ inherited blood disorder or disease‚ characterized primarily by chronic anemia and periodic episodes of pain. It is one form of sickle cell disease‚ a category of blood disorders caused by defective hemoglobin. Hemoglobin is a substance
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Agee Ms. Songer AP Biology February 16 2015 Sickle Cell Anemia: Case Study Summary Sickle Cell Anemia is a painful diseases that is caused by a mutation in the protein called hemoglobin which helps carry oxygen in red blood cells. Because of the mutation‚ the hemoglobin is shaped oddly which results in pain because it is hard for the blood to travel throughout the body and anemia because of the lack of oxygen in the blood. A person can only have this disease if both parents are carriers and they receive
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Sickle Cell Anemia first came into the view of the world around 1910 when Dr. James Herrick; a cardiologist‚ had a patient who complained of pain and described symptoms that sounded like anemia. He handed the case down to his assistant who‚ after taking a blood sample‚ discovered that the patient’s blood cells were not shaped like normal blood cells. When the patient’s blood cells were compared to normal blood cells‚ they appeared to be “sickle shaped”. After seeing this for himself Dr. Herrick took
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Sickle cell anemia was first discovered in the year of 1910. A young man by the name Walter Clement Noel from the island of Grenada‚ studied in Chicago. He went to Dr. James B. Herrick‚ whom was a cardiologist‚ with symptoms of anemia‚ who assigned Dr. Ernest Irons to the case. There Dr. Irons noticed that Noel’s red blood cells were the shape of a sickle. Although sickle cell anemia has occurred in Africa for thousands of years‚ Dr. Herrick was the first to provide a formal description of sickle
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Adams 1 Dwayne Adams Instructor: Croshaw Medical Terminology 1 18‚ April 2013 Sickle Cell Anemia Sickle-cell Anemia is a genetic blood disorder caused by the presence of an abnormal form of hemoglobin molecules in which the red blood cells loose their disc-shape and become crescent shaped. The shape also known as “hemoglobin S”. unlike normal red cells which are usually smooth and malleable‚ tend to collect after releasing oxygen‚ and cannot squeeze through small blood vessels. The
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Case Study 97 1. Sickle cell disease is a group of disorders that affects hemoglobin‚ the molecule in red blood cells that delivers oxygen to cells throughout the body. People with this disorder have atypical hemoglobin molecules called hemoglobin S‚ which can distort red blood cells into a sickle‚ or crescent‚ shape. SCD affects millions of people worldwide‚ particularly those with African‚ Spanish‚ Mediterranean‚ and Indian ancestry. Some 120‚000 infants are born with SCD every year worldwide
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Sickle cell anemia is a genetic disease that affects the shape and functionality of red blood cells. It is caused by a mutation in the DNA of the protein‚ hemoglobin‚ specifically in the beta chain. There are 531 base pairs in this DNA strand. Substitution or point mutation occurs‚ causing “GAG” to become “GTG”. This results in valine being created instead of glutamate. The mutation causes the hemoglobin to cling together in low oxygen levels and the red blood cell changes shape‚ preventing it from
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