Excess fluid volume related to inactivity‚ secondary to congestive heart failure‚ as manifested by rapid weight gain‚ pitting edema in extremities‚ elevated blood pressure‚ bilateral crackle lung sounds‚ bradypnea‚ and dyspnea. Goal: Absence of fluid retention by discharge Desired Outcomes: 1. Blood pressure within regular limits by discharge 2. Absence of edema by discharge 3. Slow progression of weight gain by day 2 of admission 4. Ease of respirations within 2 hours of admission
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puts pressure on the walls of the blood vessels (especially in the legs). This causes blood plasma to leak into the interstitial spaces. This causes one of the major signs of right sided heart failure which is pitting edema of the legs. But‚ the fluid back up does not only cause edema. The failure of the right side of the heart to load sends a smaller amount of blood to the
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nutritional needs of children‚ particularly in cases of milk allergy.[5] Kwashiorkor (pronounced /kwɑːʃiˈɔrkər/) is an acute form of childhood protein-energy malnutrition characterized by edema‚ irritability‚ anorexia‚ ulcerating dermatoses‚ and an enlarged liver with fatty infiltrates. The presence of edema caused by poor nutrition defines kwashiorkor.[1] Kwashiorkor was thought to be caused by insufficient protein consumption but with sufficient calorie intake‚ distinguishing it from marasmus
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d XIV. Vasodilator * From the Latin words "vasa‚" which means "vessel‚" and "dilate‚" which means "to make wider.“ * Work by relaxing the smooth muscles that line the walls of blood vessels -- causing the blood vessels to increase in diameter and allow blood to flow through more easily. * Vasodilators also increases plasma rennin concentration resulting in sodium and water retention but these undesirable side effects can be blocked by concominant use of a diuretic and a beta blocker.
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#3 #3 Reason for admission: High blood pressure STUDENT: Jeannette Sutton PATIENT INITIALS: HPT ROOM #_____ Nursing Diagnosis: Risk for imbalanced fluid volume r/t decreased sodium level AEB patient taking hydrochlorothiazide. Supporting data: Thiazide drugs promote excretion of Na‚ CL‚ K and water. First time pt will be taking this drug. STG: Pt will maintain sodium levels of 135-145 by 1700hrs after taking initial dose this morning. Interventions: 1. Administer medication
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excess) * Increased intracellular water; edema * Brain cell swelling‚ irritability‚ depression‚ confusion * Systemic cellular edema‚ including weakness‚ anorexia nausea‚ and diarrhea * Edema | >155 meq/L | * Excessive hypertonic salt solutions * IV hypertonic sodium * Saline-induced abortions * Selected infant formulas * Hyperaldosteronism * Cushing syndrome | Hypervolemia: * Weight gain * binding pulse * increased BP * edema * venous distentionNeuromuscular: *
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Compression therapy is a reliable method of treatment used for a variety of venous disorders and diseases‚ including varicose veins‚ deep vein thrombosis‚ and edema. As these maladies almost always occur in the lower body‚ compression stockings are the most effective and most prescribed treatment. While it is not known precisely how compression therapy works so well‚ it evidently prevents the blood from pooling and/or clotting‚ which helps the blood to flow more quickly back to the heart. Additionally
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Alterations in Fluids‚ Electrolytes‚ and Acid-Base Balance Composition & Compartment Distribution of Body Fluids ← Body fluids are distributed between the intracellular fluid (ICF) and extracellular fluid (ECF) compartments. The ICF compartment consists of fluid contained within all of the billions of cells in the body. It is the larger of the two compartments‚ containing approximately two thirds of the body water in healthy adults. ← The remaining one third of body water is in
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Diuretics Hypertension‚ Congestive Heart Failure ‚ Diabetes‚ Cirrhosis and Renal Failure Objectives I. Review renal structure and function II. Neurohumoral regulation of water (ECF) & electrolyte balance III. Pharmacotherapy that acts at the kidney A. Loop diuretics B. Thiazide diuretics C. Carbonic Anhydrase Inhibitors D. K+- sparing diuretics E. Osmotic diuretics F. Others IV. Therapeutic uses I. Structure/Function Germann and Stanfield Fig. 19.2 The Nephron: Tubular Component
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chemical messeges‚ while postsynaptic toxins affect the places nerve signals are interpreted such as muscles and other nerves. Hemotoxic venom destroys tissues in different ways. Many hemotoxins cause problems such as cardiac arrest‚ kidney failure‚ edema‚ necrosis‚ and intense pain. These effects are cause by hemotoxins anticoagulation effects. Enzymes are found commonly in both hemo-and neurotoxins‚ and enzymes play a crucial role in snake
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