SIADH case study
The primary intervention of SIADH includes fluid restriction. In case of acute episodes of SIADH, diuretics such as IV Mannitol and IV Furosemide may be given to promote diuresis and free water clearance. In severe cases, a hypertonic infusion such as 3% NaCl is administered intravenously for 36-48 hours or until the lab results shows an improved Na level. (Porth, 2009).
Physiological action and possible complications: Furosemide works by blocking the absorption of sodium, chloride, and water from the filtered fluid in the kidney tubules, causing a profound increase in the output of urine (diuresis). Common side effects of furosemide include low blood pressure, dehydration and electrolyte depletion such as hypokalemia. Less common side effects include jaundice, ringing in the ears (tinnitus), sensitivity to light (photophobia), rash, pancreatitis, nausea, diarrhea, abdominal pain, and dizziness. Increased blood sugar and uric acid levels also may occur. When hypertonic fluids are infused, osmosis pulls water out of the cells and causes the cells to shrink resulting cells to get rid of excessive free water. The side effect includes, when they shrink at the site of IV infusion, infection at the site of injection, venous thrombosis or phlebitis extending from the site of injection, extravasation and hypervolemia The primary intervention of SIADH includes fluid restriction. In case of acute episodes of SIADH, diuretics such as IV Mannitol and IV Furosemide may be given to promote diuresis and free water clearance. In severe cases, a hypertonic infusion such as 3% NaCl is administered intravenously for 36-48 hours or until the lab results shows an improved Na level. (Porth, 2009).
Physiological action and possible complications: Furosemide works by blocking the absorption of sodium, chloride, and water from the filtered fluid in the kidney tubules, causing a profound increase in the output of urine (diuresis). Common side effects of
Physiological action and possible complications: Furosemide works by blocking the absorption of sodium, chloride, and water from the filtered fluid in the kidney tubules, causing a profound increase in the output of urine (diuresis). Common side effects of furosemide include low blood pressure, dehydration and electrolyte depletion such as hypokalemia. Less common side effects include jaundice, ringing in the ears (tinnitus), sensitivity to light (photophobia), rash, pancreatitis, nausea, diarrhea, abdominal pain, and dizziness. Increased blood sugar and uric acid levels also may occur. When hypertonic fluids are infused, osmosis pulls water out of the cells and causes the cells to shrink resulting cells to get rid of excessive free water. The side effect includes, when they shrink at the site of IV infusion, infection at the site of injection, venous thrombosis or phlebitis extending from the site of injection, extravasation and hypervolemia The primary intervention of SIADH includes fluid restriction. In case of acute episodes of SIADH, diuretics such as IV Mannitol and IV Furosemide may be given to promote diuresis and free water clearance. In severe cases, a hypertonic infusion such as 3% NaCl is administered intravenously for 36-48 hours or until the lab results shows an improved Na level. (Porth, 2009).
Physiological action and possible complications: Furosemide works by blocking the absorption of sodium, chloride, and water from the filtered fluid in the kidney tubules, causing a profound increase in the output of urine (diuresis). Common side effects of