Herpesvirus-3 Research Paper

H. herpesvirus-3 is an enveloped DNA virus. H. herpesvirus-3 is an Alphaherpesvirinae.

In 1888 the relationship between chickenpox and shingles were observed by Von Bokay when children who had not acquired an immunity to H.herpesvirus-3, or have never had chickenpox, became sick with chickenpox after coming in contact with the latent H.herpesvirus-3, or someone who had shingles.
It was not until the end of the 19th century that chickenpox was reliably distinguished from small pox.

H. herpesvirus-3 can code for anti-apoptotic proteins that interferes with the apoptosis pathway. Apoptosis is the programed death of a cell once it knows that it has been infected, because H. herpesvirus-3 encodes for anti-apoptotic proteins, it is able to evade apoptosis and keep the infected cell alive therefore ensuring that its DNA can replicate within that cell without the infected cell dying.
herpesvirus-3 can cause chickenpox in a person by entering the body through one of the principle portals of entry, specifically the Respiratory tract, from the skin vesicles of an infected person. If a person has never had chickenpox and comes into contact with someone who is infected with Shingles, the first person will contract chicken pox, not shingles. This is because shingles is a latent disease and one would need to have had chickenpox before being able to get shingles. Shingles is not a communicable