"Systemic lupus erythematosus" Essays and Research Papers

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    deficiencies also increase the risk for disseminated gonococcal infection as a result of endometrial exposure of submucosal vessels to the infecting organisms. Disseminated gonococcal infection (Kerle KK et al 1992) is a rare complication‚ were a systemic spread of the bacteria in the bloodstream may cause‚ tenosynovitis‚ and dermatitis but can also present as perihepatitis.

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    Case Study

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    though the disease systemic lupus erthematosus (SLE) doesn’t have a test that specifically diagnosis this diseases‚ there are test that are run to help with understand contributing factors. The significance of a positive antinuclear antibody (ANA) titer is that 98% of people with systemic lupus test positive‚ even though it is not enough for a diagnosis of lupus (healthy individuals can test positive) it is a good indication along with other diagnostics test in the presence of lupus. The anti-double-stranded

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    revealed that D.W. had a positive antinuclear antibody (ANA) titer‚ positive dsDNA (positive lupus erythematosus)‚ positive anti-Sm (antismooth muscle antibody)‚ elevated C-reactive protein (CRP) and erythrocyte sedimentation rate (ESR)‚ and decreased C3 and C4 serum complement. Joint x-ray fi lms demonstrated joint swelling without joint erosion. D.W. was subsequently diagnosed with systemic lupus erythematosus (SLE). She was initially treated with sulindac 200 mg PO bid‚ prednisone 20 mg/day PO‚ bed

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    Ischemias Research Paper

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    TIAs are powerful forerunners of stroke. Approximately 10% of patients diagnosed as having a TIA have a stroke in the 90 days following the TIA diagnosis‚ with half of them having a stroke within 2 days of the TIA. TIAs are short-lived episodes of acute‚ focal‚ nonconvulsive neurologic dysfunction presumably caused by reversible ischemia to an area of the retina or brain. Onset of symptoms is sudden and often unprovoked‚ reaching maximum intensity almost immediately. Completed Stroke A completed

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    Autoimmune Disorders

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    pancreatic acinar cells | Ovarian Failure / Infertility | p-450 | Pernicious Anemia | Parietal cells | Ulcerative Colitis | p-ANCA | Active chronic hepatitis Addison’s disease Autoimmune atrophic gastritis AIHA Dermatomyositis Discoid lupus erythematosus Goodpasture’s syndrome Hashimoto’s thyroiditis ITP IDDM MS Myasthenia gravis Pemphigus vulgaris Pernicious anemia Primary biliary cirrhosis Primary myxedema RA Scleroderma SLE Thyrotoxicosis Classification Organ-specific

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    Your 20-year-old patient is going to start a clinical desensitization program for his allergy to pollen. 1. Describe the pathophysiologic changes to the immune system when exposed to allergens. Allergy is “a hypersensitivity response against an environmental antigen (allergen); the most common are type 1 hypersensitivities (pollen‚ molds‚ fungi‚ certain foods‚ animal dander‚ dust). Others include Type II and Type III (rare but include antibiotics and soluble antigens produced by infectious agents)

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    We have made an emergency travel to my country this July‚ which was too unexpected and cannot imagine the situation at this point. We lost our Mom (56). She had struggled with SLE (Systemic Lupus Erythematosus) since past 8 years. During this time period‚ she was regularly followed by the Doctors advice and medicine with acute care. But‚ everything effort went in vain this July. She was admitted in the hospital 25 days before this was happened. We were in regular touch about the health condition

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    what do you want

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    1. Discuss the anatomy of the middle mediastinum? middle mediastinum‚ which consists of the pericardium and heart. 2. Describe the pericardium and mention its nerve supply? Taha lecture slides 3 and 5 3. What are the structures that can be affected by cardiomegaly? the lungs and heart are occupy the mediastinum delicately. And the lung is a passive pressure sensitive organ. So the increase in heart size (usually in LV) will variably effect the lung function which will be compressed

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    disease may be primary‚ or secondary to another underlying illness. The most common causes of secondary warm-type AIHA include lymphoproliferative disorders (e.g.‚chronic lymphocytic leukemia‚ lymphoma) and other autoimmune disorders (e.g.‚ systemic lupus erythematosus‚ rheumatoid arthritis‚ scleroderma‚ ulcerative colitis) The effects are usually related to the lack of oxygen and manifest themselves in the form of weakness‚ lethargy‚ and an increase in the heart and respiration rate. Vomiting or abdominal

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    Arthritis Facts

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    Arthritis facts * Arthritis is inflammation of one or more joints. * Symptoms of arthritis include pain and limited function of joints. * Arthritis sufferers include men and women‚ children and adults. * A rheumatologist is a medical arthritis expert. * Earlier and accurate diagnosis can help to prevent irreversible damage and disability. What is arthritis? What causes arthritis? Arthritis is a joint disorder featuring inflammation. A joint is an area of the body where two different

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