Introduction Duchenne Muscular Dystrophy is a dangerous and rare disorder. It’s transferred through family generations because it’s a genetic disease. In addition‚ Duchenne Muscular Dystrophy is referred to by other names including DMD‚ Duchenne Syndrome‚ and Pseudohypertrophy. DMD is when the body cannot make dystrophin so it results in muscle weakness. Mode of Inheritance Duchenne Muscular Dystrophy is an X-linked recessive disorder and that’s why it’s more common in males. As for females‚
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Szili-Torok‚ T.‚ Theuns‚ D. J.‚ Kardos‚ A.‚ Geleijnse‚ M. L.‚ Balk‚ A. M.‚ & ... Simoons‚ M. L. (2011). Indications and Outcome of Implantable Cardioverter-Defibrillators for Primary and Secondary Prophylaxis in Patients with Noncompaction Cardiomyopathy. Journal Of Cardiovascular Electrophysiology‚ 22(8)‚ 898-904. doi:http://dx.doi.org.library.gcu.edu:2048/10.1111/j.1540-8167.2011.02015.x Edelman‚ C.‚ & Mandle‚ C. L. (2010). Health promotion throughout the life span (7th ed.). St. Louis‚
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could be coronary artery disease (CAD)‚ which is when the arteries that supply the heart with oxygen and blood‚ become blocked or narrowed. Other causes include previous heart attack due to the tissue scarring‚ or cardiomyopathy which is damage to the heart muscle (myocardium). Cardiomyopathy is caused by problems other than decreased blood flow. These causes include infections‚ alcohol‚ or drug use. Other factors that can contribute to congestive heart failure are hypertension (high blood pressure)
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for cell graft or cell-based therapy. Another research “Disease Modeling and Phenotypic Drug for Diabetic Cardiomyopathy using Human Induced Pluripotent Stem Cells” illustrates the molecular and chemical changing of human induced pluripotent stem cells in the presence of diabetic extracellular environment. This research leads us to better understand of the pathogenesis of diabetic cardiomyopathy‚ which has no specific treatment
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by avoiding going out in bright sun as much as possible‚ and use of protective clothing and high factor sunscreens must be a must. As a drug treatment corticosteroids may be used topically or intralesionally. Very potent forms are necessary for hypertrophic lesions. Fluocinonide cream may be more effective than hydrocortisone in treating people with discoid lupus erythematosus (DLE). Systemic steroids do not seem to be effective. When systemic treatment is required‚ hydroxychloroquine is the first-line
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6 degrees. Examination today reveals a child who appears in moderate distress. His skin is hot‚ and temperature was documented in the office at 39 degrees Celsius. Weight today was 51-1/2 pounds. Exam of the throat reveals the tonsils to be hypertrophic and kissing in the midline. The uvula is somewhat deviated to the left. There is erythema of the tonsils bilaterally with a peritonsillar abscess present on the right. The soft palate is bulging somewhat. Nose: The nares are patent with some clear
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adequate blood to the other organs of the body. Causes of CHF are coronary artery disease‚ past myocardial infarction‚ hypertension‚ heart valve disease‚ cardiomyopathy‚ congenital heart defects‚ endocarditis and myocarditis (American heart association‚ 2013) .In the case scenario of Mr. P 76 year old man comes with the history of cardiomyopathy and CHF and in the past repeatedly admitted for the management of CHF symptoms. This essay discusses about approach to care‚ treatment plan‚ patient and family
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Anatomy of Peripheral Nerves Peripheral nerves consist of fascicles that contain myelinated and unmyelinated axons. Endoneurium is the small amount of matrix that is present between individual axons. The perineurium is a sheath of special‚ fiber-like cells that ties the axons of each fascicle together. Epineurium is the connective tissue that surrounds the entire nerve trunk and gives off vascular connective tissue septa that traverse the nerve and separate fascicles from one another. | | Single
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cellular regeneration of heart tissues with the use of site-specific recombination to insert a normal gene to correct the defective potassium ion channel. The potassium channel causes an error in a gene that adds to an error in the protein product. Cardiomyopathy affects the heart muscles directly and become enlarged. As this disease worsens the heart becomes weaker which makes the heart less able to pump the blood through the body and maintain electrical rhythm. To repair the heart from enlarged muscles
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will progressively lose the ability to sit upright‚ walk‚ move their hands and arms‚ and breathe without difficulty. The physical limitations of DMD will eventually lead to other health problems associated with lung and heart function such as cardiomyopathy. Therefore‚ medications and therapies to manage the physical limitations of the disability must be introduced in order to decrease the progression of muscle deterioration and improve one’s quality of
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