Intravenous (IV) study day Guided independent Study In preparation for administering IV medication when you have registered as a nurse‚ it is important that you have some theoretical understanding of related issues. Completion of this guided independent study workbook will enable you to review some topic areas related to IV therapy. Once this is completed‚ you will be able to observe IV therapy in your practice placements and you will be able to take part in drawing up IV medication. NB Student
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Reinke’s Edema. Stated in Voice Disorders: Scope of Theory and Practice (Ferrand 2012) “Polyps are benign growths on the vocal fold mucosa that may result from laryngeal hyperfunction…” (p. 283). Despite the similar name‚ Polypoid Degeneration or Reinke’s Edema is not like a polyp. It is a chronic inflammatory disease in which the entire membranous portion of the vocal folds becomes infiltrated with thick‚ gelatinous fluid (Martins‚ Fabro‚ Domingues‚ Chi‚ and Gregorio‚ 2009). Reinke’s Edema is irregular
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Mount Carmel College of Nursing Columbus‚ Ohio Nursing 521: Advanced Pathophysiology CASE STUDY#1: CONGESTIVE HEART FAILURE Stephanie Barber September 21‚ 2011 INITIAL HISTORY: 66 year-old white male increasing shortness of breath over the last month noticed feet and ankles swelling by end of the day has occasional episodes of chest tightness has been waking up in the middle of the night with acute
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1. George Brown‚ 72 years of age‚ is a male patient who is admitted with the diagnosis of acute pulmonary edema secondary to acute left ventricular heart failure. The patient has a history of coronary artery disease that has been treated medically. The patient is anxious‚ pale‚ cold‚ clammy‚ and dyspneic. The vital signs are: blood pressure 88/50 mm Hg‚ heart rate 110 bpm‚ respiratory rate 32 breaths/min‚ and temperature 97°F. There are bubbling crackles and wheezing throughout the lung fields and
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was unfortunately not the case; all the dangers of Everest can lead to hypoxia‚ frostbite‚ falling into a crevasse‚ or High Altitude Pulmonary Edema (HAPE). Krakauer describes how Ngawang Topche‚ a Sherpa who assisted Scott Fischer’s Everest expedition‚ developed HAPE while climbing the mountain and the effect it had on him. High Altitude Pulmonary Edema is “typically brought on by climbing too high‚ too fast” where “the lungs fill with fluid” from the pulmonary arteries‚ which are exposed to high
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Interventions | Rationales | Evaluation:Goals met? | Risk for excess fluid volume related to inability of kidneys to excrete fluid and excessive fluid intake as evidenced by edema‚ hypertension and shortness of breathSubjectiveR.P said “ouch” when touching areas with edema (feet and legs)ObjectiveLegs and feet have edema +4‚ high blood pressure and gasps for air | One Short term R.P will be able to have a fluid balance (urine output 600-1000ml/day) within one day. | 3 Diagnostic**Monitor Respiratory
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describing compression‚ however there is very little research describing the role of elevation. Elevation is typically used in an attempt to retard edema formation. However‚ the effects of elevation in retarding edema formation following acute phase injury are nonexistent. In fact‚ the small body of research describing the effects of elevation on edema have largely used non-injured subjects. It is unclear whether elevation plays any important role in acute injury management. This has international
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hydro-alcoholicextract from aerial parts of N.officinalewas investigated in acute and sub¬¬-acute models of inflammation. Materials and methods:Different models like carageenan induced hind paw edema in rats‚ formalin induced paw edema in rats and 12-O-tetradecanoylphorbol-13-acetate (TPA)evoked ear edema in mice were used for studying the anti-inflammatory activity
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Brain edema is an increase in brain tissue water content‚ resulting in increased volume of the brain and intracranial pressure [23].In patients with intracerebral hemorrhage‚ edema is formed within a few hours after intracerebral hemorrhage‚1 to 2 weeks reach the peak[24].A Study[25] showed that‚ in the large animal model of cerebral hemorrhage (such as pigs)‚ edema around the hematoma is mainly distributed in the white matter. The formation of brain edema after intracerebral hemorrhage
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compression pump promotes increased circulation to reduce swelling and leg pain. With three modes of operation‚ including massage‚ lymphatic‚ and recovery‚ the four-chamber Vive sequential compression pump works to relieve symptoms related to venous edema‚ lymph edema‚ post-thrombotic syndrome‚ peripheral arterial disease‚ DVT‚ PE‚ and diabetes. Pressure is adjustable in a range of 30-250 mmHg for 10 ‚20 or 30 minutes for a personalized experience. The lightweight pump is portable and includes
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