I spent eight hours with the patient in the ‘Ornish’ Cardiac program. Many challenges influence the overall health of communities. Cardio vascular (CV) disease is one of the leading cause of death in the U.S. However‚ by educating the public we can bring the awareness to prevent CV diseases in our community. One of the goal of the St Jude Cardiac Rehab program to provide education to the patient‚ so they can learn how to empower their health. I spent time with a new patient during her admission process
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Nikul Patel DS13 25-12-03 Effects of exercise on cardiac output (For results see graph paper) Analysis After the initial period of rest in both girls and boys heart rates rose slightly with light exercise (walking)‚ and then fell again slightly back towards the original resting point. Then during the period of anticipation of exercise the males heart rate rose by a further 1 beat per 6 seconds‚ whereas the females took a dramatic leap‚ and rose by 3.5 beats per 6 seconds‚ or 35 beats per minute
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Compare and contrast cardiac and smooth muscle. In cardiac muscle‚ each heartbeat is triggered by the hearts own pacemaker cells‚ which initiate electrical discharge and when this reaches the contractile muscle cells (the cardiomyocytes)‚ they create an action potential which increases the concentration of calcium ions into the cell. Calcium ions play a key role in activating what is known as the contractile machinery – the actin and myosin filaments. The cardiac action potential‚ unlike other forms
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PH210 PHARMACY CLERKSHIP HOSPITAL PUTRAJAYA Case 5 Congestive Cardiac Failure Nephrotic Syndrome Community-Acquired pneumonia Siti Hasmah Bt Mohd Suffian (2006200606) CASE SUBJECT Name: MAM Gender: Male Age: 24 years old Race: Malay Weight: 138 kg BMI: 55 (morbidly obese) Height: 159cm DOA: 21st March 2010 SUBJECTIVE Chief complaint: • bilateral lower limb swelling for the past 2 weeks. • scrotal swelling for the past 3 days but noted on DOA had progressively
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Symptoms) P Decreased Cardiac Output R/T E Atrial Fibrillation and Mechanical Ventilation AEB S – Client on mechanical ventilation. Albumin 1.1 – 2/4/14 – low osmolality in blood – third spacing. Atrial Fibrilation Sluggish Pupil response Blood pressure 97/39 Heart Rate 54 Peripheral pulses diminished PLANNING ____________________________________________________________________________________ Client Goal: Client will not have decreased Cardiac Output Outcome Criteria
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Commentary Prediction and prevention of sudden cardiac arrest Heikki V. Huikuri MD See related research article by Reinier and colleagues on page 1705 and at www.cmaj.ca/lookup/doi/10.1503/cmaj.101512 Competing interests: None declared. This article was solicited and has not been peer reviewed. Correspondence to: Dr. Heikki V. Huikuri‚ heikki.huikuri@oulu.fi CMAJ 2011. DOI:10.1503 /cmaj.111245 CMAJ ajor advances have been made in understanding the causes of and treatments for cardiovascular
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Name: lily Exercise 6: Cardiovascular Physiology: Activity 1: Investigating the Refractory Period of Cardiac Muscle Lab Report Pre-lab Quiz Results You have not completed the Pre-lab Quiz. 07/10/13 page 1 Experiment Results Predict Question: Predict Question 1: When you increase the frequency of the stimulation‚ what do you think will happen to the amplitude (height) of the ventricular systole wave? Your answer : a. The amplitude will increase. Predict Question 2: If you deliver multiple
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I remember the time when I used to work‚ as a cardiac monitor technician in the Intensive care unit of a local hospital in Tampa. I was watching the patient’s monitors while one of them started showing and ST elevation on the cardiac rhythm‚ (ST elevations usually on the strip can mean different things and depending on the leads showing on the screen‚ it might represent an infarct). In that case‚ the patient was experimenting a heart attack. Suddenly‚ the patient started complaining of shortness
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This is a case study of a 76 year old female patient who is suffering from congestive cardiac failure. She has past medical history of hypertension‚ chronic renal failure‚ type 2 diabetes mellitus and hyperlipidaemia. She has been admitted in hospital several times recently and she was discharged 11 days ago from emergency department. Now she is suffering from dyspnoea and she feels like she can not catch her breath due to congestion of lungs. Congestion of lungs occurred due to congestive heart
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department‚ an ECG and cardiac enzymes were obtained. The cardiac monitor showed sinus tachycardia with occasional premature ventricular complexes. K.R. was treated with 2 L nasal oxygen‚ tissue plasminogen activator‚ sublingual nitroglycerin‚ and IV morphine sulfate. When he was pain free‚ he was transferred to the cardiac unit for monitoring. Discussion Questions 1. What changes in “cardiac enzymes” would be consistent with a diagnosis of MI? Troponins(I and T specific to cardiac muscle cells)‚ elevated
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