Risk Predictors CC and PCI Predictors of Vascular Complications Post Diagnostic Cardiac Catheterization and Percutaneous Coronary Interventions Cheryl J. P. Dumont‚ PhD(c)‚ RN‚ CCRN; Arlene W. Keeling‚ PhD‚ RN; Cheryl Bourguignon‚ PhD‚ RN; Ian J. Sarembock‚ MB‚ ChB‚ MD; Melanie Turner‚ RN‚ BSN Care of patients after cardiac catheterization and/or percutaneous coronary intervention is largely the responsibility of nurses. The identification of risk factors for vascular complications from these procedures
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The heart is composed of cardiac muscle which enable the heart to contract and allow the synchronization of the heart beat. The heart wall is divided into three layers: the epicardium‚ myocardium‚ and endocardium. * Epicardium - outer protective layer of the heart. * Myocardium - muscular middle layer wall of the heart. * Endocardium - inner layer of the heart that is continuous with the inner lining of blood * Cardiac Conduction * Cardiac conduction is the rate at which the
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Action potential‚ impulse conduction (italics)‚ and cardiac cycle (bold) Pacemaker cells in the SA node‚ found in the wall of right atrium‚ begin as polarized‚ with Na+ on the outside of the cell (sarcolemma) and the cell at its resting potential (-80 mV in the atria‚ -90 mV in the ventricles). The atria are full or filling with blood. The SA (and technically the AV) nodal cells gradually drift towards their threshold after each repolarization and depolarize (the prepotential: spontaneous depolarization)
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is the symptom of acid reflux and GERD; however‚ not everyone with acid reflux has heartburn and not everyone with heartburn has acid reflux. The symptom of heartburn can also be caused by other unusual things such as intestinal motility problems. Cardiac problems can also mimic heartburn and you should not confuse the two. Unexplained chest pain should be evaluated by an exercise stress test or EKG prior to an evaluation for gastrointestinal
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heart: the electrocardiogram Electrical activity is a basic characteristic of the heart and is the stimulus for cardiac contraction. Disturbances of electrical function are common in heart disease. Their registration as an electrocardiogram (ECG) plays an essential role in the diagnosis and management of heart disorders. THE GENESIS OF THE ELECTROCARDIOGRAM Pathways of conduction and the electrocardiogram The sinus node is situated in the right atrium close to the entrance of the
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level. In addition‚ there is a disc osteophyte complex and disc space narrowing at L5-S1 with mild spinal stenosis and left greater than right mild foraminal stenosis. Furthermore‚ there is impingement upon the left L5 nerve root. EMG and nerve conduction report dated 05/06/14 revealed evidence of bilateral L5 lumbar radiculopathy and bilateral peripheral neuropathy. Per operative reports‚ IW underwent left L4-5 and L5-S1 transforaminal ESI on 11/07/2014; 05/15/2015; and 07/24/2015‚ and left L4-5
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h. Heparin xvii. HIGH risk IV xviii. Stim. Anti throm. II i. Lovenox xix. SQ in lovehandles xx. Adv. Face swelling j. Beta-blockers xxi. 1. Decrease heart rate‚ contract.‚ and conduction xxii. 2. Constrict bronchi xxiii. Lopressor xxiv. Uses- stage fright‚ HTN‚ migraine. xxv. Adverse- Hypotension‚ brady‚ brochospasm. k. ACE inhibitors xxvi. Vasotec xxvii. Block
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Purpose The purpose of this experiment was to become familiar with the electrocardiograph to help us determine the electrical activity of the heart. As well as‚ to observe how exercise and different body positions(conditions) can affect the activity of the heart. Introduction Trained professionals can look at the EKG tracing and determine if the heart is normal or if the heart is abnormal. “An ECG records the electrical activity of the heart. The heart produces tiny electrical impulses which spread
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Most of the cardiac injury ultimately results in development of cardiac fibrosis due to negligible regenerative capacity of heart. Cardiac fibrosis is associated with increased deposition of matrix proteins in the myocardium. The expansion of the cardiac interstitial space in absence of significant cardiomyocyte loss is “reactive interstitial fibrosis” and the formation of scar in response to myocardial infarction is called “reparative fibrosis” [10‚ 46]. Increased deposition of endomysial and perimysial
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science staff (cardiac physiologist). A cardiac physiologist’s role comprises of recording and analysing a variety of physiological data required to assist in the diagnosis and treatment of different cardiac diseases (Scst.org.uk‚ 2016). They are qualified to perform a range of non-invasive cardiovascular procedures‚ from electrocardiograms to pacemaker and ICD checks which I have observed during placement. In addition‚ cardiac physiologists are also trained to assist in cardiac catheterization
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