Warfarin in Atrial Fibrilation in Different Age Subgroups Title and abstract Item 1: Title The purpose of this study is to perform cost-effectiveness analysis (CEA) to determine the cost effectiveness of Diabigatran Etexilate. In this study‚ the researcher studied about Dabigatran Etexilate as an intervention and compared its effectiveness for stroke and systemic embolism risk reduction with Warfarin which is a comparator in patients of different age group with atrial fibrillation. Item 2:
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left ventricle is restricted and left atrial pressure rises‚ pulmonary venous congestion Poor lung compliance and breathlessness. There is dilatation and hypertrophy of the left atrium‚ left ventricular filling becomes more dependent on left atrial contraction. Exercise‚ pregnancy Requires increased cardiac output Increase in heart rate Diastole shortens when mitral valve is open Further rise in left atrial pressure The mitral valve orifice
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Case Study 1Question: What is atrial fibrillation‚ how is this different from normal rhythm? Answer: Atrial fibrillation is a rapid chaotic rhythm in the upper chamber of the heart. It is different than normal rhythm because normally the heart sinus node during normal rhythm; the upper chamber contracts and then the lower chamber contracts. When you develop atrial fibrillation the upper chamber suddenly starts beating at rapid rates -- 300 to 600 beats a minute‚ in a very chaotic fashion‚ and that
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are called the atria and the bottom chambers‚ the ventricles. A normal heart beat begins when the Sino-Atrial Node‚ located on the posterior wall of the right atrium. The Sino-Atrial Node initiates the "wave of contraction" which results in the contraction of the atria. The Atrio-Ventricular Node delays the signal initiates by the Sino-Atrial node for about 0.15 seconds‚ permitting full atrial systole before ventricular systole. This ensures that the ventricles are filled before the ventricles contract
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infarction. • Normal sinus rhythm starts in the sinoatrial (SA) node and follows the normal conduction pattern of the cardiac cycle. o The P wave represents the depolarization of the atria (passage of an electrical impulse through the atria)‚ causing atrial contraction. o The PR interval represents the time period for the impulse to spread through the atria‚ atrioventricular (AV) node‚ bundle of His‚ and Purkinje fibers. o The QRS complex represents depolarization of the ventricles (ventricular contraction)
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Atrial Fibrillation and the clinical impact Atrial Fibrillation (AFib) is most frequent cardiac arrhythmias that characterized by extremely rapid irregular atrial rhythm; resulted from ectopic focal impulses production. An estimated prevalence of 33.5 million people suffering from AFib with the incidence of 5 million new cases annually. Associate with the substantial clinical complications like heart failure‚ embolus such as cerebral emboli with a portion of 25%-30% of all acute ischemic stroke
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comes into the cardiology office‚ complaining of fatigue‚ palpitations‚ shortness of breath with mild activity‚ ongoing consistently for the past week without any chest pain or syncopal episodes. She is a 75-year-old woman with a history of atrial fibrillation (AFib)‚ controlled type 2 diabetes‚ myocardial infarction (MI) with a history of percutaneous coronary intervention to right coronary artery‚ stage IV chronic renal failure‚ and a mild mitral regurgitation on ECHO 7 years ago. Her in clinic
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What are the characteristics of ventricular fibrillation? 2. Describe the changes in atrial pressure‚ ventricular pressure‚ aortic pressure and ventricular volume that occur during the various stages of the cardiac cycle. Illustrate when the various valves are open or closed. 3. In order for both of the semilunar valves
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Most of the cardiac injury ultimately results in development of cardiac fibrosis due to negligible regenerative capacity of heart. Cardiac fibrosis is associated with increased deposition of matrix proteins in the myocardium. The expansion of the cardiac interstitial space in absence of significant cardiomyocyte loss is “reactive interstitial fibrosis” and the formation of scar in response to myocardial infarction is called “reparative fibrosis” [10‚ 46]. Increased deposition of endomysial and perimysial
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inferior-Posterior infarction The above ECG changes can be found if there is an occlusion of the: RCA LAD circumflex all of the above. the most complications associated with this problem is ventricular dysrhythmias. AV block. atrial flutter. hemodynamic changes and dysrhythmia caused by SA node & AV node. Identify the Rhythm in the below figure Unifocal Bigeminy. Multifocal trigeminy Unifocal Trigeminy Multifocal PVCs. The nurse would be most concerned
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