The traditional phenylketonuria (PKU) is an example of inborn errors in metabolism and this is categorized by a total lack of an enzyme called phenylalanine hydroxylase‚ this enzyme usually converts phenylalanine to tyrosine‚ http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1684467/ if this conversion doesn’t take place in causes the blood to build up with phenylalanine causing a problem in the urinary tracks. The genetic disorder causes mutilation of brain development‚ follow-on with mental retardation
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Matthew Lee Bio 10 - Study Guide 2 CELLULAR DIVISION * What is a chromosome‚ chromatid‚ chromatin?chromosome is a thread like structure of nucleic acids holding information. Chromatin super coil material part of strand. Chromatid is one strand of the chromatin. * How do they change after S phase? Makes 2 chromatids * What are the stages of interphase? What happens in each stage?G1-cell growth‚S phase-Synthesis‚G2 prepare for division * What are the stages of mitosis? What happens
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Christopher Scott Biol 241 homework Professor: Greg Crowther 1. Compare and contrast the reasons cell division is important for unicellular and multicellular organisms. Cell division is the method in which single celled organisms reproduce. Cell division allows multi-cellular to grow and repair dead or damaged cells 2. Provide an example of why cell division remains important to an adult organism even after it isfully developed. Some cells divide to heal wounds such as cuts or broken bones.
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AP Biology Mid-Term Study Guide 2015 Monitha Patel Chemical Bonding Compounds Sharing of a pair of valence electrons by two or more different atoms Molecule Two or more atoms held together by covalent bonds Ionic Electron attraction between two atoms Cations (+1) and Anions (-1) attract Covalent Sharing of valance electrons between atoms Nonpolar Electrons are shared equally O2 Polar Covalent Unequal sharing of electrons between atoms Two hydrogen atoms are bonded to a single oxygen
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SCI206 Unit 3 Individual Project American Intercontinental University SCI206-1001A-13 Abstract This assignment discusses the relationships between reproduction‚ heredity and DNA. Genetics – From Genes to Proteins‚ Mutations During transcription‚ the information in the DNA of a specific gene is copied into mRNA (messenger RNA)‚ which creates a nucleotide sequence. After transcription‚ if the DNA base sequence is 3’-TACCCTTTAGTAGCCACT-5’‚ then the base sequence of mRNA would be 5 ’-AUGGGAAAUCAUCGGUGA-3’
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|cross over | |dominant |frequency of recombination | |recessive |map unit | |allele |marker | |homozygote/homozygous |mapping function | |heterozygote/heterozygous
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Evolution‚ Genes & the Environment Variation is when you refer to genes; it means that there is a large variety of phenotypes‚ alleles and genotypes which can sometimes cause genetic variation. This results in different physical characteristics. Some examples of variations in humans are eye colour‚ hair colour and gender. Variations caused by the environment are the climate (tanned/pale skin) this is affected by the environment as the sun comes in contact with your skin and will darken it‚ the
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Quiz #1 (Pay attention to the answer key) Question 1 In the scientific method‚ the condition which is deliberately changed is referred to as the D. experimental variable. Question 2 Which of the following is not a step in the scientific method? D. Scientific Law Question 3 When an organism’s DNA is permanently altered‚ we say that it E. mutates. Question 4 Which answer choice is not a defining characteristic of life? C. disorder
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| c | C | C | Cc | CC | c | cc | Cc | C= normal allele‚ dominant c= allele for cystic fibrosis Genetics: 1 out of 4 children will be double recessive (homozygous)25% 2 out of 4 children will have cystic fibrosis trait (heterozygous)50% 1 out of 4 children will be homozygous dominant 25% There is 75% chance that their children will have the allele for cystic fibrosis. There is a 25% chance their children won’t have the allele for cystic fibrosis. Cystic fibrosis is
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As the common question has been asked before‚ where did the blue budgie color morphing first take place? The first appearance of the blue budgie color morphing first took place in 1878 in Brussels [1]‚ then later reappeared dominantly in the Netherlands in the early 1880’s. Although the first color mutation was in the early 1880s‚ reproduction of the blue budgie gene was very unpredictable as scientists have documented. The first results in breeding the new blue budgies resulted in short life spans
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