Unit 1 Case Study 1

Topics: Red blood cell, Bone marrow, Blood, Stem cell, Immune system, White blood cell / Pages: 2 (347 words) / Published: Jan 7th, 2015
Unit 1 Case Study 1: Tell Me About Blood
Stephanie Sprague
ITT Technical Institute

Unit 1 Case Study 1: Tell Me About Blood
1)What is the significance of a lower-than-normal hematocrit? What is the effect of a bacterial infection of the hematocrit? Can cause bleeding destruction of red blood cells causing sickle cell anemia and an enlarged spleen it can also cause decreased production of red blood cells which in turn can cause cancer and bone marrow suppression. Nutritional problems as well as over hydration are caused by lower-than-normal hematocrit levels. Bacterial infection may lower your hematocrit levels.
2)Compare the development of lymphocytes with the development of the other formed elements. Lymphocytes and other formed elements are developed from pluripotent stem cells. The pluripotent stem cells generate myeloid stem cells and lymphoid stem cells. Lymphoid stem cells begin development in the red bone marrow, but some are completed in the lymphatic tissue, where they give rise to lymphocytes. Myeloid stem cell begins and complete their development in the red bone marrow and give rise to red blood cells, platelets, basophils, monocytes, neutrophils, and eosinophils.
3)What is erythropoiesis? Which factors speed up and slow down erythropoiesis? Erythropoiesis is a process in which red blood cells are produced. It is stimulated by decreased O2 in circulation, which is detected by the kidneys which then secrete the hormone erythropoietin, increased level of physical activity can also cause an increase in erythropoiesis. It slows down when there is sufficient oxygen carrying capacity of the blood.
4)Explain what would happen if a person with type B blood were given a transfusion of type O blood. Nothing would happen type O is a universal donor.
5)
References
Li, W., Wang, J., Long, R., Su, G., Bukhory, D., Dai, J., & ... Wang, Z. (2014). Novel Antibody against a Glutamic Acid-Rich Human Fibrinogen-Like Protein 2-Derived Peptide near Ser91 Inhibits hfgl2



References: Li, W., Wang, J., Long, R., Su, G., Bukhory, D., Dai, J., & ... Wang, Z. (2014). Novel Antibody against a Glutamic Acid-Rich Human Fibrinogen-Like Protein 2-Derived Peptide near Ser91 Inhibits hfgl2 Prothrombinase Activity. Plos ONE, 9(4), 1-13. doi:10.1371/journal.pone.0094551 Sreenivasamurthy, S. K., Dey, G., Ramu, M., Kumar, M., Gupta, M. K., Mohanty, A. K., & ... Keshava Prasad, T. S. (2013). A compendium of molecules involved in vector-pathogen interactions pertaining to malaria. Malaria Journal, 12(1), 1-7. doi:10.1186/1475

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