Traumatic Head Injury Case Studies
INTRODUCTION
The clinical scenario given is a serious one. Therefore, extreme care and caution will be required in assessing the patient and managing any injuries discovered in the process. In this essay, the potential clinical problems the patient could have would be explores, and based on the clinical parameters provided, attempt would be made to interpret and subsequently manage the signs, symptoms as well as the injuries presented by the patient.
In order to identify quality diagnostic methods, recommendations and evidenced-based management plan, search and review of existing guidelines addressing traumatic head injury was carried out. Also Current books and texts that was relevant to traumatic brain injuries which also included recommendations …show more content…
(2005). Contusion in the head commonly indicates a degree of bleeding beneath the skull layer Li et al. (2009) and in this case, external bleeding is not ruled out. The other potential injuries include cranial indentation, intracranial lesion and possible skull fracture Silvestri and Aronson, (1997). The aetiology of these injuries depends on the force of impact of the accelerating or decelerating cricket ball, Sanders et al. (2010). Furthermore, the relationship between skull fracture and raised intracranial pressure following traumatic head injury has been widely investigated by Matthew et al. (2007). The conclusion of their empirical studies was a direct correlation. Consequently, there is a risk of raised intra cranial pressure, (ICP) in this patient. McMillian and Roger, (2009) result also corroborates Gregory and Ward, (2010) which list head trauma as one of the causes of ICP. In this scenario, the aetiology would be that the force of impacting ball compromised the protective layer of the delicate brain organs (cerebral cortex, cerebellum and the brain stem) Sanders, et al. (2010) this may rupture, lacerate or burst blood vessels in any of these brain organ Li, et al. (2009). This situation potentially could lead to increase in extrastitial fluid contents of the brain with little or insufficient outlet causing a fatal rise in ICP Clifton,
The clinical scenario given is a serious one. Therefore, extreme care and caution will be required in assessing the patient and managing any injuries discovered in the process. In this essay, the potential clinical problems the patient could have would be explores, and based on the clinical parameters provided, attempt would be made to interpret and subsequently manage the signs, symptoms as well as the injuries presented by the patient.
In order to identify quality diagnostic methods, recommendations and evidenced-based management plan, search and review of existing guidelines addressing traumatic head injury was carried out. Also Current books and texts that was relevant to traumatic brain injuries which also included recommendations …show more content…
(2005). Contusion in the head commonly indicates a degree of bleeding beneath the skull layer Li et al. (2009) and in this case, external bleeding is not ruled out. The other potential injuries include cranial indentation, intracranial lesion and possible skull fracture Silvestri and Aronson, (1997). The aetiology of these injuries depends on the force of impact of the accelerating or decelerating cricket ball, Sanders et al. (2010). Furthermore, the relationship between skull fracture and raised intracranial pressure following traumatic head injury has been widely investigated by Matthew et al. (2007). The conclusion of their empirical studies was a direct correlation. Consequently, there is a risk of raised intra cranial pressure, (ICP) in this patient. McMillian and Roger, (2009) result also corroborates Gregory and Ward, (2010) which list head trauma as one of the causes of ICP. In this scenario, the aetiology would be that the force of impacting ball compromised the protective layer of the delicate brain organs (cerebral cortex, cerebellum and the brain stem) Sanders, et al. (2010) this may rupture, lacerate or burst blood vessels in any of these brain organ Li, et al. (2009). This situation potentially could lead to increase in extrastitial fluid contents of the brain with little or insufficient outlet causing a fatal rise in ICP Clifton,