Skeletal Muscle Report
The muscle physiology of skeletal muscle was observed by using electrical, physical, and neural stimulations of an isolated gastrocnemius muscle from Rana pipiens. The gastrocnemius receives signals from the action potentials of the sciatic nerve. The muscle contraction is caused by the binding of a neurotransmitter once the action potential reaches the neuromuscular junction. Stimulation of the muscle and the sciatic nerve allows for recording and measuring of these properties. We observed twitch recruitment of the nerve and muscle length-tension relationship of the skeletal muscle, summation of skeletal muscle contractions, tetanus, skeletal muscle fatigue, and the effects of temperature on skeletal muscle contractions. Since motor neutrons
…show more content…
At a frequency of 1 Hz, the peaks started out with an average amplitude of 1.166133N, and the troughs started out with an average amplitude of 1.68933N (Fig. 4). As the frequency increased, so did the peak and trough size until tetanus was reached at 20Hz: peak amplitude 1.611933 + 0.015366N and trough amplitude 1.611933 + 0.015366N (Fig. 4). There was significant difference observed between the trough and peaks contraction amplitudes before tetanus. There was no significant difference observed between the trough and peaks contraction amplitudes during …show more content…
Varying Temperature: In the nerve for cold temperate the maximum the contraction 0.7607 N, and the contraction threshold was observed at 130 mV (Fig. 5A). In the room temperature, the maximum contraction amplitude was 0.7065 N, and the contraction threshold was observed at 150 mV (Fig. 5A) The warm temperature maximum contraction amplitude was 0.6180 N, and the contraction threshold was observed at 100mV (Fig. 5A).
In the muscle, for cold temperature Ringer’s solution, the maximum contraction was 0.4121N and, the contraction threshold was observed at 550 mV (Fig. 5B) The room temperature the maximum contraction amplitude was 0.3106N, and the contraction threshold was observed at 350 mV (Fig. 5B). The warm temperature maximum contraction amplitude was 0.2867 N, and the contraction threshold was observed at 350mV (Fig.
At a frequency of 1 Hz, the peaks started out with an average amplitude of 1.166133N, and the troughs started out with an average amplitude of 1.68933N (Fig. 4). As the frequency increased, so did the peak and trough size until tetanus was reached at 20Hz: peak amplitude 1.611933 + 0.015366N and trough amplitude 1.611933 + 0.015366N (Fig. 4). There was significant difference observed between the trough and peaks contraction amplitudes before tetanus. There was no significant difference observed between the trough and peaks contraction amplitudes during …show more content…
Varying Temperature: In the nerve for cold temperate the maximum the contraction 0.7607 N, and the contraction threshold was observed at 130 mV (Fig. 5A). In the room temperature, the maximum contraction amplitude was 0.7065 N, and the contraction threshold was observed at 150 mV (Fig. 5A) The warm temperature maximum contraction amplitude was 0.6180 N, and the contraction threshold was observed at 100mV (Fig. 5A).
In the muscle, for cold temperature Ringer’s solution, the maximum contraction was 0.4121N and, the contraction threshold was observed at 550 mV (Fig. 5B) The room temperature the maximum contraction amplitude was 0.3106N, and the contraction threshold was observed at 350 mV (Fig. 5B). The warm temperature maximum contraction amplitude was 0.2867 N, and the contraction threshold was observed at 350mV (Fig.