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Patho 2

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Patho 2
1. Judy has fair skin / red hair, her skin has less melanin than Mariah’s. This puts her at higher risk for melanoma than Mariah.
2. Judy observed that her mole was larger and more jagged than she remembered. It was discolored, with a raised purplish-black dot. This occurred because cells called melanocytes produce more pigment. Discolored or multicolored moles are considered atypical.

1. Benign tumors have no potential to spread, and will not affect the organ’s function. However removal of malignant tumors can prove more complicated, as organs can be affected by its presence / lack of presence. Another problem of treating malignant tumors is that they spread to other parts of the body, called metastasis. Benign tumors don't metastasize; they stay in one place, simply getting bigger and bigger. Once they're removed, you don't have to worry about treating a recurrence

2. Almost any common injury or infection would cause your body to produce more cells.
3. We all have cycle proto-oncogenes because in normal, healthy cells they are what regulate cell growth and division. A person with a mutated form of the oncogene is at an increased risk for developing cancer because cell growth is not going to be normal. The mutated oncogenes interfere with the "timer" that tells a cell not to divide anymore. The reason that not everyone has cancer is because while everyone has the oncogenes, not everyone has a mutated form of them.

1. Genetics, smoking, UV rays, and chemicals increase a person’s chances of having melanoma.
2. UVA radiation damages the DNA in human melanocyte cells, causing mutations that can lead to melanoma.
3. Predisposition means a close member of your family has cancer you have a high probability of developing cancer. It takes more than a mutated cell cycle gene to automatically get cancer, however you are in fact predisposed to cancer. Engaging in risky behaviors such as sunbathing still does not guarantee cancer, however increases your

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