Sexual Response in Cardiovascular Disease

Topics: Sexual intercourse, Blood, Human sexuality Pages: 2 (475 words) Published: September 26, 2011
Sexual Response in Cardiovascular Disease
By G. Jackson, Journal of Sex Research (2009, May)
46(2/3), 233-236
Basic Anatomy and Physiology
October 6, 2011
Heidi Peterson

Sexual Response in Cardiovascular Disease

Disease amongst men and women vary in degrees, but also share a thread of commonality between the two sexes. Atherosclerosis, better known as CAD-Coronary Artery Disease is a build-up of plaque within the lumen of the vessel wall causing narrowing or stenosis of the vein or artery; eventually leading to cardiovascular disease. Cardiovascular disease is the largest cause in the established population today of morbidity and mortality. More often than not, women are not the forefront of this disease, and are innately more threatened by the thought of breast cancer being the culprit of death and not thinking twice regarding cardiovascular disease. (Jackson, 2009) Most research articles focus on men with erectile dysfunction or a sexual problem that inhibits normal intercourse and a cardiac event transpiring, when in all actuality; men and women with or without sexual dysfunction will be at risk related to the presence of cardiovascular disease. (Jackson, 2009)

Sexual intercourse as an “activity” mimics mild to moderate physical exercise performed on a daily basis. Research performed using ambulatory electrocardiography (ECG) and blood pressure monitoring, compared heart rate, electrocardiographic, and blood pressure responses during sexual activity and other activities of daily living. (Jackson, 2009) Two positions were recorded, the man being on top and beneath the women. In either position, the peak heart rate was 114 [ or -] 14 beats per minute returning to 69 [ or -] 12 beats per minute by 120sec post orgasm with the man on top; and ranging to 117 [ or -] 4 beats per minute. Blood pressure readings remained steady in both positions, not fluctuating deviant of 160mg Hg. (Jackson, 2009) Final monitoring of stable angina...
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