DISSEMINATED HERPES ZOSTER
Submitted to Professor Kelly Hampton Nadeau, MN, RN
In partial fulfillment of the requirements Nursing 305
Description of PATHOPHYSIOLOGY DISORDER
Herpes Zoster or simply zoster is so known as Zona, People who suffer from Herpes zoster endure painful[->0] skin rashes with blisters[->1], sometimes in a limited area on one side of the body. The Chicken Pox virus suffered by young Children and young Adults is caused from the initial infection of Herpes Zoster. People with Herpes Zoster and Herpes simplex, because of the similarity between the two names and due to the fact that they are both caused by the Varicella Zoster Virus. When people have outbreaks from the Varicella Zoster virus, the rash heals within two or four weeks, but by the virus spreading from one or ganglia along the nerves of an affected segment, People usually experience pain from nerve damage usually for months or years. Natural History of Herpes Zoster (2007)
Etiology of the disorder
Herpes Zoster is caused by the same virus that causes Chicken Pox. When people have the Chicken Pox viruses from their younger years, the virus can sometimes stay inactive, living in nerve roots and become active causing “Singles”, (Another name for Herpes Zoster) in one’s adult years. People become immune to the virus after having the Chicken Pox or Chicken Pox vaccine, having a weakened immune system, or stress. Epidemiology of the disorder
Herpes Zoster infect people worldwide, it’s know to have a worldwide prevalence. The disease can only occur in people who previously had the Chicken Pox. There is a strong relationship with People with Herpes Zoster and increasing age, there’s no relationship to season or epidemics. The incidence rate of herpes zoster ranges from 2 to 3 cases per 1,000 person-years among healthy individuals, increasing to 3.9–11.8 per 1,000 person‐years among those older than 65 years, and incidence rates worldwide are similar. This relationship with age has been demonstrated in many different countries, and is attributed to the fact that cellular immunity declines as people grow older. Weinburg, JM (2007) Its pathophysiology on all body systems
Herpes Zoster is common to manifest in 1 or more posterior spinal ganglia or cranial sensory ganglia. The reason being is viral particles have been preserved within these ganglia in a
dormant state since the original episode of varicella. As a result, there’s pain and characteristic pertaining to its findings along with corresponding sensory dermatomes of the involved ganglia. Some involvement of the anterior and posterior horn cells, pia-arachnoid, and peripheral nerves is observed, with consequent muscle weakness or palsy, pleocytosis of spinal fluid, and/or sensory loss. Rarely, inflammation of the spinal cord, membranes, brain, or visceral involvement may occur. Gnann JW Jr, Whitley RJ (2002) Its clinical signs/symptoms and why they occur
According to Kotani N, Kushikata T, Hashimoto H (2000), Varicella Zoster Virus is the same virus that causes chickenpox as well as the virus that cause Herpes Zoster (Shingles). It occurs in individuals of who already had chickenpox. Shingles are more common in adults aging 50 years and older but can also occur in people of all ages. There are early signs of herpes zoster which include a feeling of uneasiness, discomfort, headaches and fevers. There are additional symptoms that follow depending on the dermatome involved. Shingles begins with sensations that are described by burning, itching, oversensitivity, or tingling on one side of the body.
Usually, when a fever occurs in one to two days a rash of blisters follows and appears in a pattern. There is pain from the shingles which can be mild to severe depending upon the part of the skin that’s affected. Within a few days the shingle blisters becomes ulcers or open sores in which can...
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