Buergers Dse
BUERGER’S DISEASE
A nursing case study
Presented by: Group 26
Charlotte faith Valeroso
Barbie Joy Tumaliuan
Chramaigne Tumaru
Rovierose Sotelo
Leslie Agngarayngay
Grace Tabanda
Rohmar jones Tingonong
Caroline D. UY
Submitted to:
Ms. Divina L. Malana, RN, MSN
INTRODUCTION
Buerger’s disease is thrombotic and inflammatory occlusion of small arteries and veins among smokers. It is also known as ThromboangiitisObliterans. It involves inflammation and fibrosis of nerves. It results in thrombus formation and segmental occlusion of the vessels. It is differentiated from other vessel diseases by its microscopic appearance. In contrast to atherosclerosis, Buerger’sdisease is believed to be an autoimmune vasculitis that results in occlusion of distal vessels.
Buerger’s disease occurs most often in men between 20 and 35 of age, and it has been reported in all races and in many areas of the world. There is considerable evidence that heavy smoking or chewing tobacco is a causative or an aggravating factor.
The clinical manifestations are pain, foot cramps, especially of the arch (instep claudication), after exercise and cold sensitivity. Physical signs include intense rubor (reddish- blue discoloration) of the foot and absence of pedal pulse, but with normal femoral and popliteal pulses. If the upper extremities are involved, the radial and ulnar artery pulses are absent or diminished. As the disease progresses, definite redness or cyanosis of the part appears when the extremity is in a dependent position. Color changes may progress to ulceration, and ulceration with gangrene eventually occurs.
Segmental limb blood pressures are taken to demonstrate the distal location of the lesions or occlusions. Duplex ultrasonography is to document patency of the proximal vessels and to visualize the extent of distal disease. Contrast angiography is used to identify the diseased portion of the anatomy. Treatment focuses on improving circulation to the extremities,
A nursing case study
Presented by: Group 26
Charlotte faith Valeroso
Barbie Joy Tumaliuan
Chramaigne Tumaru
Rovierose Sotelo
Leslie Agngarayngay
Grace Tabanda
Rohmar jones Tingonong
Caroline D. UY
Submitted to:
Ms. Divina L. Malana, RN, MSN
INTRODUCTION
Buerger’s disease is thrombotic and inflammatory occlusion of small arteries and veins among smokers. It is also known as ThromboangiitisObliterans. It involves inflammation and fibrosis of nerves. It results in thrombus formation and segmental occlusion of the vessels. It is differentiated from other vessel diseases by its microscopic appearance. In contrast to atherosclerosis, Buerger’sdisease is believed to be an autoimmune vasculitis that results in occlusion of distal vessels.
Buerger’s disease occurs most often in men between 20 and 35 of age, and it has been reported in all races and in many areas of the world. There is considerable evidence that heavy smoking or chewing tobacco is a causative or an aggravating factor.
The clinical manifestations are pain, foot cramps, especially of the arch (instep claudication), after exercise and cold sensitivity. Physical signs include intense rubor (reddish- blue discoloration) of the foot and absence of pedal pulse, but with normal femoral and popliteal pulses. If the upper extremities are involved, the radial and ulnar artery pulses are absent or diminished. As the disease progresses, definite redness or cyanosis of the part appears when the extremity is in a dependent position. Color changes may progress to ulceration, and ulceration with gangrene eventually occurs.
Segmental limb blood pressures are taken to demonstrate the distal location of the lesions or occlusions. Duplex ultrasonography is to document patency of the proximal vessels and to visualize the extent of distal disease. Contrast angiography is used to identify the diseased portion of the anatomy. Treatment focuses on improving circulation to the extremities,